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Insects in food…
March 3, 2023 In The Talk News
Insects found in JUNK FOOD
https://pubmed.ncbi.nlm.nih.gov/33640239/
2) Much like shellfish allergies, chitin allergies are an anaphylaxis risk. Not reading labels of food stuffs containing insect protein can prove to be a huge problem. Even if not allergic, it can induce lung inflammation.
3) in addition to inflammation, when you block one of the major therapeutics that treat inflammation, that could be disastrous.
4) In countries and populations naive to this component in their diet, will there be dose response severity? Who wants to roll the dice? By binding to Toll-like receptor 2 (TLR-2) it triggers inflammation.
5) We are carnivorous. Their goal is to take away meat, under the completely false guise of #FakeAGW aka anthropogenic climate change. Truth is, it’s easier to rule a weaker population. That said, will they start sneaking insect protein in our foods? Nothing fancy, just crickets
6) Read your labels and when you find it, share it on social media to alert others of this potentially harmful additive. Any ingredient that came from something with an exoskeleton should be labeled with an allergy warning.
7) and H/T to on evidence if prion rods in some ectopods. That’s a risk I’m not too interested in taking.
Nigel Farage warns new EU law classifying insects as FOOD will have huge impact on UK – ‘It’s time for a proper Brexit’…
https://www.gbnews.uk/news/nigel-farage-warns-new-eu-law-classifying-insects-as-food-will-have-huge-impact-on-uk-its-time-for-a-proper-brexit/432312 How is it with the imported food from EU, the food will have crickets in it? They will put crickets’ powder in the food the send us to America?
In The Talk News
https://inthetalknews.com/2023/01/27/its-official-european-food-will-now-contain-crickets/
Who wants this to eat and not knowing?
THANKS TO: https://inthetalknews.com/2023/03/03/insect-in-food/
Chitin induces steroid-resistant airway inflammation and airway hyperresponsiveness in mice
Affiliations
Free article
Methods: We developed mouse models of inhaled chitin particle-induced airway inflammation and steroid-resistant ovalbumin (OVA)-induced airway inflammation. Some experimental groups of mice were treated additionally with dexamethasone (DEX). Murine alveolar macrophages (AMs), which were purified from bronchoalveolar lavage (BAL) fluids, were incubated with chitin, and treated with or without DEX.
Results: The numbers of total cells, AMs, lymphocytes, eosinophils, and neutrophils among BAL-derived cells, as well as the IL-1β levels in BAL fluids and the numbers of IL-1β-positive cells in lung, were significantly increased by chitin stimulation. Airway hyperresponsiveness (AHR) was aggravated in mice of the chitin inflammation model compared to control animals. The production of IL-1β was significantly increased in murine AMs by chitin treatment, but DEX administration did not inhibit this chitin-induced IL-1β production. Furthermore, in mouse models, DEX treatment inhibited the OVA-induced airway inflammation and AHR but not the airway inflammation and AHR induced by chitin or the combination of OVA and chitin.
Conclusions: These results suggest that inhaled chitin induces airway inflammation, AHR, and the production of IL-1β. Furthermore, our findings demonstrate for the first time that inhaled chitin induces steroid-resistant airway inflammation and AHR. Inhaled chitin may contribute to features of steroid-resistant asthma.
Keywords: Airway inflammation; Alveolar macrophages; Chitin; Interleukin-1β; Steroid-resistant asthma.
Copyright
2021 Japanese Society of Allergology. Production and hosting by Elsevier B.V. All rights reserved.
See all similar articles
CONTINUE HERE: https://pubmed.ncbi.nlm.nih.gov/33640239/
March 3, 2023 In The Talk News
Insects found in JUNK FOOD
https://pubmed.ncbi.nlm.nih.gov/33640239/
2) Much like shellfish allergies, chitin allergies are an anaphylaxis risk. Not reading labels of food stuffs containing insect protein can prove to be a huge problem. Even if not allergic, it can induce lung inflammation.
3) in addition to inflammation, when you block one of the major therapeutics that treat inflammation, that could be disastrous.
4) In countries and populations naive to this component in their diet, will there be dose response severity? Who wants to roll the dice? By binding to Toll-like receptor 2 (TLR-2) it triggers inflammation.
5) We are carnivorous. Their goal is to take away meat, under the completely false guise of #FakeAGW aka anthropogenic climate change. Truth is, it’s easier to rule a weaker population. That said, will they start sneaking insect protein in our foods? Nothing fancy, just crickets
6) Read your labels and when you find it, share it on social media to alert others of this potentially harmful additive. Any ingredient that came from something with an exoskeleton should be labeled with an allergy warning.
7) and H/T to on evidence if prion rods in some ectopods. That’s a risk I’m not too interested in taking.
Nigel Farage warns new EU law classifying insects as FOOD will have huge impact on UK – ‘It’s time for a proper Brexit’…
https://www.gbnews.uk/news/nigel-farage-warns-new-eu-law-classifying-insects-as-food-will-have-huge-impact-on-uk-its-time-for-a-proper-brexit/432312 How is it with the imported food from EU, the food will have crickets in it? They will put crickets’ powder in the food the send us to America?
In The Talk Newshttps://inthetalknews.com/2023/01/27/its-official-european-food-will-now-contain-crickets/
Who wants this to eat and not knowing?
THANKS TO: https://inthetalknews.com/2023/03/03/insect-in-food/
Chitin induces steroid-resistant airway inflammation and airway hyperresponsiveness in mice
Affiliations
- PMID: 33640239
- DOI: 10.1016/j.alit.2020.12.004
Free article
Abstract
Background: Previous reports have shown that pathogen-associated patterns (PAMPs) induce the production of interleukin (IL)-1β in macrophages. Moreover, studies using mouse models also suggest that chitin, which acts as a PAMP, induces adjuvant effects and eosinophilic infiltration in the lung. Thus, we investigated the effects of inhaled chitin in mouse models.Methods: We developed mouse models of inhaled chitin particle-induced airway inflammation and steroid-resistant ovalbumin (OVA)-induced airway inflammation. Some experimental groups of mice were treated additionally with dexamethasone (DEX). Murine alveolar macrophages (AMs), which were purified from bronchoalveolar lavage (BAL) fluids, were incubated with chitin, and treated with or without DEX.
Results: The numbers of total cells, AMs, lymphocytes, eosinophils, and neutrophils among BAL-derived cells, as well as the IL-1β levels in BAL fluids and the numbers of IL-1β-positive cells in lung, were significantly increased by chitin stimulation. Airway hyperresponsiveness (AHR) was aggravated in mice of the chitin inflammation model compared to control animals. The production of IL-1β was significantly increased in murine AMs by chitin treatment, but DEX administration did not inhibit this chitin-induced IL-1β production. Furthermore, in mouse models, DEX treatment inhibited the OVA-induced airway inflammation and AHR but not the airway inflammation and AHR induced by chitin or the combination of OVA and chitin.
Conclusions: These results suggest that inhaled chitin induces airway inflammation, AHR, and the production of IL-1β. Furthermore, our findings demonstrate for the first time that inhaled chitin induces steroid-resistant airway inflammation and AHR. Inhaled chitin may contribute to features of steroid-resistant asthma.
Keywords: Airway inflammation; Alveolar macrophages; Chitin; Interleukin-1β; Steroid-resistant asthma.
Copyright
2021 Japanese Society of Allergology. Production and hosting by Elsevier B.V. All rights reserved. Similar articles
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See all similar articles
CONTINUE HERE: https://pubmed.ncbi.nlm.nih.gov/33640239/
